Week 4 Discussion – Due

Feb 4, 2021 11:59 PM

NSG6002-Health Policy and Health Promotion in Advanced Nursing Practice CP04

Describe what specific resources or federal agencies that you will utilize to remain informed and current in public health trends and practices

Submission Details:

• Post your response to the Discussion Area by the due date assigned. Respond to at least two posts by the end of the week.

Week 4 Discussion

Discussion Topic

Due February 4 at 11:59 PM

Week 4 Project

Assignment

Due February 8 at 11:59 PM

 Health Policy Paper

Forward the topic of the health policy paper to faculty at the end of Week One,

Health policy unfolds daily and drives clinical practice in the US. The student will investigate current policies or legislation underway for a specific health-related issue. The Student will develop a scholarly APA formatted paper supported by evidence. The policy paper rubric:

Introduction to population or problem (incidence, prevalence, epidemiology, cost burden etc)

Description of how the policy is intended for a specific population, program or organization

Specific legislators involved in the policy development and dissemination

Identify the role of the APRN in assisting with the policy or refuting the policy – this requires the evidence to support opinion, ideas and/or concepts.

Discuss how the policy influences clinical practice and is used to promote best outcomes

Examine how the policy can be used by the interprofessional team to ensure coordinated and comprehensive care for the specific population

Conclusion – summarize findings

APA format – use of primary peer-reviewed references as much as possible

Submission Details:

• Support your responses with examples.
• Cite any sources in APA format.
• Submit your document to the Submissions Area by the due date assigned.

Patricia, Let’s expand your points a bit and add readings from this week…There are may not be any symptoms in patients with prostatitis or symptoms that force the patient to seek emergent care. The symptoms include: 1) frequent urge to urinate, 2) difficulty urinating, 3) dysuria, and 4) fever and chills.  What other symptoms may present?  Web MD (2005) Prostatitis. Retrieved (2012). From Article Link: http://men.webmd.com/guide/prostatitis

• Follow up question from order #SYH31938
•  
• Rhonda Johnston3 hours agoPatricia, Accurate  post! I just want to make a point about the importance of having an in-depth understanding of the pathological processes we are trying to apply in this course. If you can drill down to the cellular level instead of a broad stroke of the process, your diagnosis and treatment plan will be more accurate. There is a certain combination of clinical signs and symptoms that present depending on the cellular and chemical reactions occurring in the disease process.  One step of the inflammation process is the vascular permeability that occurs with the initial insult. This results in the swelling. In the abdominal system this could present with a tender rotund abdomen or diarrhea because of the body’s attempt to rid itself of the excess fluid. How would you treat this?  I am not so concerned in this course with the treatment, but your ability to drill down and connect the clinical symptoms with the pathological process. To expand your thoughts a bit more. One of the objectives this week was to examine the two main types of gall bladder diseases, cholelithiasis, and cholecystitis.  Cholelithiasis, as you know, are otherwise known as gallstones are hard, pebble-like formations that develop in the gallbladder.  Cholecystitis can be chronic and acute. It is defined as an inflammation of the gallbladder.  Other gallbladder diseases to consider are polyps, which are a growth of tissue in the gallbladder and do not forget cancer tumors.  Abnormal growths on the gallbladder and bile ducts are common cancers and must be included in your differential diagnosis.  The same symptoms occurring with the above can also be found in biliary dyskinesia. This is a condition where bile does not flow from the gallbladder or common bile duct and backs up in the gallbladder. How would you differentiate the causes when the symptoms are the same?   Heuman, D. (2012)  Cholelithiasis  retrieved 2012 from http://emedicine.medscape.com/article/175667-overviewReply

Week 8 Discussion

Discussion Topic

Due January 21 at 10:59 PM

Discussion Assignment

This discussion assignment provides a forum for discussing relevant topics for this week based on the course competencies covered.

For this course, you are automatically divided into discussion groups of 3 or 4 students. This is done through the course registration process. You may or may not be with the same group each week.

Instructions:

Read the Discussion Prompt Below, then post your responses/answers as indicated by the Discussion Board Prompt.

Initial Post: Post your initial writing by Day 3 of the week.*Response #1. Due by Day 5 of the week. Response #2: Due by Day 7 of the week. Response to faculty: Respond to any comments left by your faculty by end of day on Day 7 of the week.

*You will not be able to see the postings from your peers until you have made your initial post.

Start reviewing and responding to the postings of your classmates as early in the week as possible. Respond to all of the initial postings from your peers in your discussion group on different days of the week.

Every post in the discussion must be supported by evidence. Cite sources in your responses to other classmates. In addition, you must respond to any responses received from your professor. Complete your participation for this assignment by end of the week.

To support your work, use your course and text readings as well as the South University Online Library. As in all assignments, cite your sources in your work and provide references for the citations in APA format. Patient or lay person medical information portals such as webMD, Medscape, Mayo Clinic, or any disease foundations, such as the arthritis foundation or the diabetic foundation, are not acceptable resources for your scholarly work.

Discussion Prompt

Initial Post: Explain what happens physiologically with chronic renal failure and the GFR. Support with evidence. Include important labs that are monitored in the process

Please respond to my peer post below .

Post/Response #1 Respond to one of your peers in your discussion group. Identify the additional workup that is needed to rule in or rule out these differential diagnoses. What clinical signs/symptoms would you expect to see with these two differential diagnoses?

Anemia

Contains unread posts

Lorena Randazzo posted Jan 4, 2021 3:55 PM

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Anemia is a disease commonly seen in everyday medical practice, its occurrence accounts for around 2 to 4 billion cases globally due to vitamin deficiencies, chronic diseases, infections, and hemoglobin disorders among others. It reduces the number of red blood cells and hemoglobin jeopardizing the body’s oxygenation compromising several organs ( Jansen, 2019).

As per the present case scenario about a 50 years old patient presenting with fatigue, pallor, dyspnea on exertion, and palpitations, and a laboratory report of a low hematocrit 32, hemoglobin 9.7, and reticulocyte count low at 47% MCV of 78 and MCHC of 32.1. The individual s/s is due to a decrease in circulating oxygen that affects the heart, arteries, capillaries, and ventricles because the drop in RBC’s generates as well a decrease in blood volume that is compensated by the movement of interstitial fluid into the intravascular space expanding plasma. Thus, the heart cardiac output is affected to respond to the increased in metabolic needs which can lead to heart failure. In the pulmonary system, this increased demand generates an increase in the rate and depth of respiration where more oxygen is released generating shortness of breath, fatigue, dizziness (McCance & Huether,2018). 

Proper diagnostic testing results vital; the patient’s hemoglobin (hgb) is low, as well as the mean corpuscular volume (MCV), and MCHC. Hence to further determine the etiology of this problem additional biomarkers will be checked: A complete CBC including, hemoglobin and hematocrit to determine the number or circulating blood cells. Iron in conjunction with transferrin, soluble transferrin receptors ( to differentiate the type of anemia), transferrin saturation, Mentzer index ( to differentiate between iron deficiency and beta-thalassemia), ferritin, vitamin B12, to assess for iron deficiency; reticulocytes ( chronic anemia), hepcidin-25, bioactive ( hereditary anemia, iron deficiency by the liver), hemoglobin electrophoresis ( suspicion of microcytic anemia) and mean corpuscular hemoglobin (MCH) to determine hemoglobin mass per RBC (Jansen, 2019).

References

Jansen, V. (2019). Diagnosis of anemia—A synoptic overview and practical approach. Transfusion and Apheresis Science, 58(4), 375–385. https://doi.org/10.1016/j.transci.2019.06.012

McCance K., & Huether S. (2018). Pathophysiology: The Biologic Basis for Disease in Adults and Children (8th ed.). Mosby.

follow up question from order # SYH31937

Patricia, great post. Let’s add some of the readings this week and expand your points. Through the years there have been many discussion, research and new treatments for asthma. However the exact cause of asthma is still not known. Researchers think some genetic and environmental factors interact to cause asthma, most often early in life. An inherited tendency to develop allergies, called atopy may be the most related diagnostic cause. If asthma or atopy runs in the patient’s family, exposure to irritants might make their airways more reactive to substances in the air are still theories. Some other factors might be more likely to cause asthma in certain people than in others. So why can we not find the cause? U.S. Department of Health & Human Services: National Institutes of Health (2012). How Is Asthma

Treated and Controlled retrieved from http://www.nhlbi.nih.gov/health/health-topics/topics/asthma/treatment.html  

Discussion Prompt

Respiratory

Initial Post: Both bronchiolitis and bronchiectasis present with a cough and dyspnea. Describe the pathophysiologic process of both bronchiolitis and bronchiectasis. Compare and contrast the two processes.

Follow up question from order # SYH31823

• Rhonda Johnstonyesterday at 3:27 PMPatricia, The key take home this week is to be able to identify the pathological processes in all of its specificity. Then we take the process and identify what parts and pieces are not working and identify the clinical s/s and then the appropriate treatment. It is this pathway that you are to begin to understand. This is why it is important to be able to breakdown the pathological process as it will be key to how you treat your patientYou post is accurate and included many key points. Let’s add a bit to your post….The chronically high levels of circulating neuroendocrine hormones such as catecholamines, renin, angiotensin, and aldosterone affects the myocardium directly, causing structural remodeling of the heart over the long. term. Many of these remodeling effects seem to be mediated by transforming growth factor. This results in a reduction of perfusion to the skeletal muscle causes atrophy of the muscle fibers. This can result in weakness, increased fatigueability and decreased peak strength – all contributing to exercise intolerance. What tests would you see ordered to diagnosis this pathological process? What pharmacological intervention would you use in this situation? Norris, T. L. (2019). Porth’s Pathophysiology: Concepts of altered health states (9thed.) Wolters Kluwer, Philadelphia. PA.Reply

Week 4 Discussion – Due

Dec 24, 2020 10:59 PM

NSG5003-Advanced Pathophysiology CP02

Discussion Assignment

This discussion assignment provides a forum for discussing relevant topics for this week based on the course competencies covered.

For this course, you are automatically divided into discussion groups of 3 or 4 students. This is done through the course registration process. You may or may not be with the same group each week.

Instructions:

Read the Discussion Prompt Below, then post your responses/answers as indicated by the Discussion Board Prompt.Initial Post: Post your initial writing by Day 3 of the week.*Response #1. Due by Day 5 of the week. Response #2: Due by Day 7 of the week. Response to faculty: respond to any comments left by your faculty by end of day on Day 7 of the week. 

*You will not be able to see the postings from your peers until you have made your initial post.

Start reviewing and responding to the postings of your classmates as early in the week as possible. Respond to all of the initial postings from your peers in your discussion group on different days of the week.

Every post in the discussion must be supported by evidence. Cite sources in your responses to other classmates. In addition, you must respond to any responses received from your professor. Complete your participation for this assignment by end of the week.

To support your work, use your course and text readings as well as the South University Online Library. As in all assignments, cite your sources in your work and provide references for the citations in APA format. Patient or lay person medical information portals such as webMD, Medscape, Mayo Clinic, or any disease foundations, such as the arthritis foundation or the diabetic foundation, are not acceptable resources for your scholarly work.

Discussion Prompt

order #1: Initial Post: Describe the physiology of blood pressure control. Causes of Primary Hypertension may include overactivity of the SNS; overactivity of the RAAS; alterations in other neurohumoral mediators of blood volume and vasomotor tone such as ANP, BNP, and adrenomedullin; inflammation; a complex interaction involving insulin resistance and endothelial function; and obesity-related hormonal changes

Select 3 of the above potential causes and explain the pathophysiologic mechanisms of primary hypertension.

Order #2: Response #1 Post a response to one peer in your discussion group. In the post identify the organ damage that can occur as a result of Hypertension. Describe the pathophysiologic process of the organ damage.

Order# 3: Response #2 Orthostatic hypotension can occur as an adverse effect of medications used to manage hypertension. Go back to your initial post and describe the pathophysiologic process that causes orthostatic hypotension.

PLEASE ANSWER THE FOLLOWING QUESTIONS ON A SEPARATE SHEET OF ORDER. I ORDER 3 PAGES FOR THIS DISCUSSION. THE INITIAL DISCUSSION IS ORDER #1 THAN ORDER 2 AND 3 ARE THE RESPONDS 

Hello this is the first and second discussion response  from order #SYH31582.

Please answer each question on a page. 1 page per question . please used the south university library. i order 2 pages. but separate discussions per page plz

Response #1: If this were about Cystic Fibrosis, how would the responses be different to the questions in the Initial Post? Give a detailed explanation supporting your response.

Response # 2 Down Syndrome is another genetic disorder. Why is the Punnett Square not a good tool for predicting if offspring will have Down Syndrome? Why does the risk for Down’s Syndrome increase with maternal age?

Follow up question from order # SYH31565. Response #2: Go back into your own initial post.

• Identify what diagnostic testing or additional information you might need to establish a diagnosis.
• Identify what you believe is the correct diagnosis for the patient in the case study with the rationale for your response.
• Identify why the differential diagnoses left by your peers are or are not the correct diagnosis and support this with evidence from research and from the case posted by your peers.

Week 3 Discussion

Discussion Topic

Due December 17 at 10:59 PM

Discussion Assignment

This discussion assignment provides a forum for discussing relevant topics for this week based on the course competencies covered.

For this course, you are automatically divided into discussion groups of 3 or 4 students. This is done through the course registration process. You may or may not be with the same group each week.

Instructions:

Read the Discussion Prompt Below and then post your responses/answers as indicated by the Discussion Board Prompt.Initial Post: post your initial writing by Day 3 of the week.*Response #1. Due by Day 5 of the week.Response #2: Due by Day 7 of the week.Response to faculty: Respond to any comments left by your faculty by end of day on Day 7 of the week.

*You will not be able to see the postings from your peers until you have made your initial post.

Start reviewing and responding to the postings of your classmates as early in the week as possible. Respond to all of the initial postings from your peers in your discussion group on different days of the week.

Every post in the discussion must be supported by evidence. Cite sources in your responses to other classmates. In addition, you must respond to any responses received from your professor. Complete your participation for this assignment by end of the week.

To support your work, use your course and text readings as well as the South University Online Library. As in all assignments, cite your sources in your work and provide references for the citations in APA format. Patient or lay person medical information portals such as webMD, Medscape, Mayo Clinic, or any disease foundations, such as the arthritis foundation or the diabetic foundation, are not acceptable resources for your scholarly work.

Discussion Prompt

Describe the Punnett Square and what it used to predict. What are the limitations of the Punnett Square?

Mom is a carrier for hemophilia but does not have the disease. Dad does not have hemophilia

Using the Punnett Square, answer the following questions:

• What is the probability a female offspring developing hemophilia?
• What is the probability of a female offspring being a carrier for hemophilia?
• What is the probability of a male offspring developing hemophilia?
• What is the probability of a male offspring being a carrier developing hemophilia?
• What is the probability of any offspring developing hemophilia?
• Explain why only some offspring would develop the disease?

Response #1: If this were about Cystic Fibrosis, how would the responses be different to the questions in the Initial Post? Give a detailed explanation supporting your response.

Response # 2 Down Syndrome is another genetic disorder. Why is the Punnett Square not a good tool for predicting if offspring will have Down Syndrome? Why does the risk for Down’s Syndrome increase with maternal age?

Week 2 Discussion

Discussion Topic

Due December 10 at 10:59 PM

Discussion Assignment

This discussion assignment provides a forum for discussing relevant topics for this week based on the course competencies covered.

For this course, you are automatically divided into discussion groups of 3 or 4 students. This is done through the course registration process. You may or may not be with the same group each week.

Instructions:

Read the Discussion Prompt Below, then post your responses/answers as indicated by the Discussion Board Prompt.

Initial Post: post your initial writing by Day 3 of the week.*Response #1. Due by Day 5 of the week.Response #2: Due by Day 7 of the week.Response to faculty: Respond to any comments left by your faculty by end of day on Day 7 of the week. 

*You will not be able to see the postings from your peers until you have made your initial post.

Start reviewing and responding to the postings of your classmates as early in the week as possible. Respond to all of the initial postings from your peers in your discussion group on different days of the week.

Every post in the discussion must be supported by evidence. Cite sources in your responses to other classmates. In addition, you must respond to any responses received from your professor. Complete your participation for this assignment by end of the week.

To support your work, use your course and text readings as well as the South University Online Library. As in all assignments, cite your sources in your work and provide references for the citations in APA format. Patient or lay person medical information portals such as webMD, Medscape, Mayo Clinic, or any disease foundations, such as the arthritis foundation or the diabetic foundation, are not acceptable resources for your scholarly work.

Discussion Prompt

Case StudyAnna Sanchez, a 21-year-old nursing student, comes to her nurse practitioner in December with a 5-week history of itchy eyes and nasal congestion with watery nasal discharge. She also complains of a “tickling” cough, especially at night, and she has had episodes of repetitive sneezing. She gets frequent “colds” every spring and fall.

Physical Examination

• Vital Signs: Afebrile; respiratory rate, pulse, and blood pressure all normal
• Skin: Flaking erythematous rash on the flexor surfaces of both arms
• Head, Eyes, Ears, Nose, and Throat: Tender over maxillary sinuses; sclera red and slightly swollen with frequent tearing; outer nares with red irritated skin; internal nares with red, boggy, moist mucosa and one medium-sized polyp on each side; pharynx slightly erythematous with clear postnasal drainage (NOTE: Nasal polyps are common in allergic rhinitis. They are edematous protrusions of the mucosa that are infiltrated with neutrophils, eosinophils, and plasma cells.)
• Lungs: Clear to auscultation and percussion

Initial Post: Answer the following questions about Anna Sanchez and her condition.

1. What evidence suggests that Anna does not have an acute severe infection?
2. If Anna has allergic rhinitis, what type of hypersensitivity reaction is involved?
3. A skin test indicates that Anna is allergic to cat dander. Two months ago, Anna’s roommate brought home a cat. Why didn’t Anna’s symptoms start when the cat entered the household, rather than 3 weeks later?
4. What class of antibodies bind to the mast cells?
5. What physiological mechanisms caused the redness of Anna’s nasal mucosa?
6. What mechanisms caused Anna’s clear postnasal drainage?

Response # 1: Carefully read the initial posts made by the other students in your small group.

• Respond to the initial post of one of your peers in your discussion group. Identify 1 differential diagnosis for the case study presented. Identify the underlying pathophysiology and clinical presentation of the Differential Diagnosis you identified in your response to your peers. Please Note: you may not duplicate a differential diagnosis posted by another peer in the discussion thread.

Response #2: Go back into your own initial post.

• Identify what diagnostic testing or additional information you might need to establish a diagnosis.
• Identify what you believe is the correct diagnosis for the patient in the case study with the rationale for your response.
• Identify why the differential diagnoses left by your peers are or are not the correct diagnosis and support this with evidence from research and from the case posted by your peers.